 Anna M. Selmecki, PH.D.
| Title | Research Fellow in Pediatrics (EXT) |
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| Institution | Children's Hospital Boston |
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| Department | Pediatrics |
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| Address | Dana-Farber Cancer Institute
Mayer Bldg - Rm 624
44 Binney St
Boston MA 02115
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| Phone | 617/623-4343 |
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 Bibliographic
Publications listed below are automatically derived from MEDLINE/PubMed and other sources, which might result in incorrect or missing publications.
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Andaluz E, Bellido A, Gómez-Raja J, Selmecki A, Bouchonville K, Calderone R, Berman J, Larriba G. Rad52 function prevents chromosome loss and truncation in Candida albicans. Mol Microbiol. 2011 Mar; 79(6):1462-82.
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Selmecki A, Forche A, Berman J. Genomic plasticity of the human fungal pathogen Candida albicans. Eukaryot Cell. 2010 Jul; 9(7):991-1008.
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Zacchi LF, Selmecki AM, Berman J, Davis DA. Low dosage of histone H4 leads to growth defects and morphological changes in Candida albicans. PLoS One. 2010; 5(5):e10629.
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Selmecki AM, Dulmage K, Cowen LE, Anderson JB, Berman J. Acquisition of aneuploidy provides increased fitness during the evolution of antifungal drug resistance. PLoS Genet. 2009 Oct; 5(10):e1000705.
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Bouchonville K, Forche A, Tang KE, Selmecki A, Berman J. Aneuploid chromosomes are highly unstable during DNA transformation of Candida albicans. Eukaryot Cell. 2009 Oct; 8(10):1554-66.
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Forche A, Magee PT, Selmecki A, Berman J, May G. Evolution in Candida albicans populations during a single passage through a mouse host. Genetics. 2009 Jul; 182(3):799-811.
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Ketel C, Wang HS, McClellan M, Bouchonville K, Selmecki A, Lahav T, Gerami-Nejad M, Berman J. Neocentromeres form efficiently at multiple possible loci in Candida albicans. PLoS Genet. 2009 Mar; 5(3):e1000400.
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Selmecki A, Gerami-Nejad M, Paulson C, Forche A, Berman J. An isochromosome confers drug resistance in vivo by amplification of two genes, ERG11 and TAC1. Mol Microbiol. 2008 May; 68(3):624-41.
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Legrand M, Forche A, Selmecki A, Chan C, Kirkpatrick DT, Berman J. Haplotype mapping of a diploid non-meiotic organism using existing and induced aneuploidies. PLoS Genet. 2008 Jan; 4(1):e1.
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Coste A, Selmecki A, Forche A, Diogo D, Bougnoux ME, d'Enfert C, Berman J, Sanglard D. Genotypic evolution of azole resistance mechanisms in sequential Candida albicans isolates. Eukaryot Cell. 2007 Oct; 6(10):1889-904.
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Selmecki A, Forche A, Berman J. Aneuploidy and isochromosome formation in drug-resistant Candida albicans. Science. 2006 Jul 21; 313(5785):367-70.
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Coste A, Turner V, Ischer F, Morschhäuser J, Forche A, Selmecki A, Berman J, Bille J, Sanglard D. A mutation in Tac1p, a transcription factor regulating CDR1 and CDR2, is coupled with loss of heterozygosity at chromosome 5 to mediate antifungal resistance in Candida albicans. Genetics. 2006 Apr; 172(4):2139-56.
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Selmecki A, Bergmann S, Berman J. Comparative genome hybridization reveals widespread aneuploidy in Candida albicans laboratory strains. Mol Microbiol. 2005 Mar; 55(5):1553-65.
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